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For research and educational purposes only. Not intended for human consumption.

MOTS-c

Extensively Studied
Updated Dec 2025

Mitochondrial Open Reading Frame of the 12S rRNA-c | Metabolic Peptide

Injectable
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5-15mg
1x daily or 3x weekly
Injectable
Subcutaneous: abdomen
4-12 weeks
Typical duration
2-8°C
Refrigerated

Overview

What is MOTS-c?

MOTS-c is a 16-amino acid mitochondrial-derived peptide encoded by the mitochondrial genome. It acts as a mitohormone that regulates metabolic homeostasis, enhances insulin sensitivity, and promotes longevity. 2024-2025 research shows MOTS-c prevents pancreatic islet cell senescence, delays diabetes progression, and mimics exercise by binding casein kinase 2 (CK2) in skeletal muscle. MOTS-c levels decline with age, correlating with poorer metabolic health. Banned by WADA

Key Benefits

Enhanced insulin sensitivity, improved glucose metabolism through AMPK activation, exercise-mimetic effects (boosts glucose uptake, fat oxidation without exercise), prevents pancreatic islet senescence (2025), potential longevity benefits. Levels decline with age but remain stable in long-lived species like naked mole-rats.

Mechanism of Action

MOTS-c operates through the Folate-AICAR-AMPK pathway. 2024 research confirms it binds casein kinase 2 (CK2) in skeletal muscle to boost glucose uptake and fat oxidation. Under metabolic stress, it translocates to the nucleus and binds stress-response transcription factors (NRF2, ATF1/ATF7). Effects are akin to metformin.

Molecular Information

2,174.6 Da
Weight
16
amino acids
Mitochondrial-derived peptide (MDP)
Type
Amino Acid Sequence:
Met-Arg-Trp-Gln-Glu-Met-Gly-Tyr-Ile-Phe-Tyr-Pro-Arg-Lys-Leu-Arg
* Encoded by mitochondrial DNA (MT-RNR1 gene) within the 12S rRNA

Pharmacokinetics

Peak
1 hr
Half-life
4 hrs
Cleared
~20 hrs
100%50%0%0h6h12h18h24h
Peak
Half-life
Cleared
Lee et al. 2015

Research Indications

Insulin Resistance

Improves insulin sensitivity by ~30% in animal studies through AMPK activation. Enhances glucose uptake in skeletal muscle.

Type 2 Diabetes

Clinical trials show improved glucose homeostasis. Restores metabolic function through enhanced insulin receptor sensitization.

Obesity Prevention

Prevents obesity despite identical caloric intake in animal models. Promotes fatty acid oxidation and thermogenesis.

Research Protocols

Disclaimer: MOTS-c is banned by WADA as an AMPK activator. These are commonly discussed research protocols. Consult a healthcare provider before use.

GoalDoseFrequencyRoute
Metabolic health5-10mgOnce dailySubcutaneous
Anti-aging protocol15mg3x weeklySubcutaneous
Exercise performance10-15mgPre-workoutSubcutaneous
Conservative start5mgOnce dailySubcutaneous

Timing: Optimal timing is morning before exercise. Exercise enhances endogenous MOTS-c production by 11.9-fold in muscle tissue. AMPK activation occurs within 30 minutes of administration.

Peptide Interactions

How to Reconstitute

Important: Always use bacteriostatic water (BAC). Sterile technique is essential.

1

Allow vial to reach room temperature (15-20 minutes)

2

Calculate required BAC water volume using calculator

3

Draw BAC water into syringe

4

Inject slowly down vial side (not directly onto powder)

5

Gently swirl until dissolved (never shake vigorously)

6

Store reconstituted solution at 2-8°C and use within 14 days

Dosing Calculator

Calculate your injection volume with visual dosing guide

FINAL CONCENTRATION
2.50mg/mL
Each milliliter contains 2.50 mg of peptide
VISUAL REFERENCE (RESEARCH USE ONLY)

To obtain 250 mcg from this solution:

Draw 0.10 mL=10 units

(1 mL = 100 units on any insulin syringe)

0102030405060708090100
00.10.20.30.40.50.60.70.80.91.0
0.10 mL
10 units

Draw to this mark for 250 mcg

This calculator is for research purposes only. Always verify calculations and consult protocols.

Quality Indicators

White to Off-White Powder

Lyophilized powder should appear uniform without discoloration.

Clear Solution After Reconstitution

Should be colorless and clear without particles or cloudiness.

Sequence Verification

Confirm peptide sequence with Certificate of Analysis showing purity >95%.

Limited Human Data

Clinical safety data is limited. Start with conservative doses.

No FDA Approval

Not FDA-approved for human therapeutic use.

WADA Banned Substance

Prohibited by World Anti-Doping Agency as AMPK activator.

What to Expect

  • Week 1-2: AMPK pathway activation, initial glucose tolerance improvements
  • Week 2-4: Enhanced exercise capacity, improved insulin sensitivity
  • Week 4-8: Sustained metabolic benefits, potential body composition improvements
  • Week 8-12: Maximum mitochondrial function enhancement, metabolic flexibility
  • Side effects: Generally mild, possible injection site reactions

Side Effects & Safety

Side Effects

  • Generally well-tolerated in animal studies with minimal side effects
  • Monitor blood glucose if using diabetes medications - may require dose adjustments
  • Limited long-term human safety data - use with appropriate medical supervision
  • May cause mild injection site reactions (redness, swelling)
  • Not recommended during pregnancy or breastfeeding
  • Prohibited by WADA for competitive athletes (metabolic modulator)

When to Stop

  • Severe blood glucose changes
  • Allergic reactions
  • Persistent injection site issues
  • Before athletic competition (WADA)
  • Consult healthcare provider

References

5 Studies

Pancreatic Islet Senescence Prevention (2025)

Animal/Preclinical | MOTS-c administration | Delays diabetes progression

2025 study showed MOTS-c prevents pancreatic islet cell senescence, delaying diabetes progression. Effects on metabolite profiles (reduced purine/dipeptide metabolism, increased acylcarnitine/methionine cycle) similar to metformin.

View Study

CK2 Binding & Exercise-Mimetic Effects (2024)

Animal | Skeletal muscle analysis | Exercise-like metabolic benefits

2024 study confirmed MOTS-c mimics exercise by binding casein kinase 2 (CK2) in skeletal muscle, boosting glucose uptake, fat oxidation, and insulin sensitivity without actual exercise.

Type 2 Diabetes Rat Model (2024)

Animal | T2D rat model | Restored mitochondrial function

In T2D rats, MOTS-c restored mitochondrial respiration, increased mitochondrial content via AMPK pathway activation, improved glucose handling, reduced weight gain, and reversed cardiac hypertrophy.

Quick Start Guide

Typical Dose
5-15mg
How Often
Once daily or 3x weekly, preferably morning
Where to Inject
Subcutaneous: abdomen
Timing
Morning before exercise or with first meal
Effects Timeline
AMPK activation within 30 min, glucose improvements 1-2 weeks, full benefits 4-8 weeks
Storage
Lyophilized: room temp 3 weeks or freezer. Reconstituted: 2-8°C for 14 days
Cycle Length
4-12 weeks continuous or 3x weekly intermittent
Break Between
2-4 weeks between continuous cycles

Research Disclaimer

MOTS-c is sold for laboratory research purposes only and is not intended for human or animal consumption. The information provided on this page is compiled from published research, veterinary studies, and anecdotal reports for educational purposes. This content does not constitute medical advice, diagnosis, or treatment recommendations. Any research involving MOTS-c must comply with all applicable local, state, and federal regulations. BioInfinity Lab makes no claims regarding the safety or efficacy of MOTS-c for any purpose. Consult qualified professionals for any research applications.