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For research and educational purposes only. Not intended for human consumption.

SHLP-2

Limited Research
Updated Dec 2025

Small Humanin-Like Peptide 2 | Metabolic Regulation

Injectable
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2 mg/kg (mouse studies)
Research protocols vary
Injectable
IP or ICV (research)
Research-dependent
Typical duration
2-8°C
Refrigerated

Overview

What is SHLP-2?

SHLP-2 (Small Humanin-Like Peptide 2) is a mitochondria-derived peptide that provides metabolic benefits by protecting against diet-induced obesity, improving insulin sensitivity, suppressing food intake, enhancing energy expenditure, and regulating lipid metabolism in preclinical studies. It activates the CXCR7 receptor to mediate its metabolic effects.

Key Benefits

Anti-obesity effects, improved insulin sensitivity, appetite suppression, enhanced energy expenditure, lipid metabolism regulation.

Mechanism of Action

Binds to and activates chemokine receptor 7 (CXCR7). Suppresses food intake, reduces hypothalamic expression of orexigenic neuropeptides (AgRP and NPY), increases energy expenditure via enhanced thermogenesis, and activates POMC neurons in the hypothalamic arcuate nucleus.

Molecular Information

~2,500 Da (estimated)
Weight
22
amino acids
Mitochondrial-derived peptide (MDP)
Type
Amino Acid Sequence:
Met-Gly-Val-Lys-Phe-Phe-Thr-Leu-Ser-Thr-Arg-Phe-Phe-Pro-Ser-Val-Gln-Arg-Ala-Val-Pro-Leu
* Part of the humanin-like peptide family encoded by mitochondrial genome

Pharmacokinetics

Peak
Unknown
Half-life
Limited data
Cleared
Limited data
100%50%0%0h6h12h18h24h
Peak
Half-life
Cleared
Preclinical research

Research Indications

Anti-Obesity Effects

Prevents high-fat diet-induced body weight gain, reduces fat mass, and decreases hepatic steatosis in mouse models.

Appetite Suppression

Suppresses food intake for up to 8 hours post-refeeding by reducing orexigenic neuropeptide expression.

Energy Expenditure

Increases energy expenditure via enhanced thermogenesis.

Research Protocols

Disclaimer: No human clinical dosing established. All research is preclinical using mouse models. Human trials are lacking.

GoalDoseFrequencyRoute
Mouse Anti-Obesity Study2 mg/kgIP administrationIntraperitoneal
Central Administration (Research)ICV dosingAs per study designIntracerebroventricular

Timing: No established timing. Mouse studies show effects lasting up to 8 hours post-administration.

Peptide Interactions

How to Reconstitute

Important: Always use bacteriostatic water (BAC). Sterile technique is essential.

1

Allow vial to reach room temperature

2

Add appropriate sterile water or BAC water

3

Gently swirl to dissolve

4

Solution should be clear

5

Store refrigerated at 2-8°C

6

Use per research protocol guidelines

Dosing Calculator

Calculate your injection volume with visual dosing guide

FINAL CONCENTRATION
2.50mg/mL
Each milliliter contains 2.50 mg of peptide
VISUAL REFERENCE (RESEARCH USE ONLY)

To obtain 250 mcg from this solution:

Draw 0.10 mL=10 units

(1 mL = 100 units on any insulin syringe)

0102030405060708090100
00.10.20.30.40.50.60.70.80.91.0
0.10 mL
10 units

Draw to this mark for 250 mcg

This calculator is for research purposes only. Always verify calculations and consult protocols.

Quality Indicators

Preclinical Research Only

No human clinical trials. All data from mouse models and in vitro studies.

Strong Preclinical Evidence

Multiple studies showing metabolic benefits in diet-induced obesity models.

Identified Receptor Target

CXCR7 receptor identified as mechanism of action.

Age-Related Decline

Circulating levels decrease with aging, suggesting therapeutic potential.

Limited Availability

Research-grade compound with limited commercial availability.

What to Expect

  • No human timeline established
  • Mouse studies show rapid appetite suppression effects (up to 8 hours)
  • Anti-obesity effects observed over multi-week treatment periods
  • Improved insulin sensitivity and lipid profiles in preclinical models
  • Effects may vary between species

Side Effects & Safety

Side Effects

  • Limited safety data - preclinical research only
  • No serious adverse events reported in mouse studies
  • Endogenous peptide family with expected physiological safety profile
  • Monitor for any unexpected effects
  • Not approved for human use

When to Stop

  • Any unexpected adverse effects
  • Signs of metabolic disturbance
  • As directed by research protocol
  • Consult healthcare provider

References

3 Studies

Anti-Obesity Effects in HFD Mice (2023)

Mouse | 2 mg/kg IP | Multiple weeks | Prevented weight gain and hepatic steatosis

SHLP2 administration prevented HFD-induced body weight gain, reduced fat mass, lowered leptin levels, and decreased hepatic steatosis in mice.

View Study

CXCR7 Receptor Identification (2023)

High-throughput screening | In vitro | CXCR7 identified as receptor

SHLP2 binds to and activates chemokine receptor 7 (CXCR7), mediating its metabolic effects.

Ceramide Metabolism Study (2025)

Mouse | Various | Lipidomics | Lowers ceramides and sphingolipids

SHLP2 lowers plasma levels of ceramides, glycosylated ceramides, sphingolipids, and diacylglycerols.

Quick Start Guide

Typical Dose
2 mg/kg (mouse studies) - no human dose established
How Often
Research protocols vary
Where to Inject
IP or ICV in research settings
Timing
No established timing protocol
Effects Timeline
Appetite suppression within hours, metabolic effects over weeks in mice
Storage
Refrigerate at 2-8°C
Cycle Length
Research-dependent
Break Between
Not established

Research Disclaimer

SHLP-2 is sold for laboratory research purposes only and is not intended for human or animal consumption. The information provided on this page is compiled from published research, veterinary studies, and anecdotal reports for educational purposes. This content does not constitute medical advice, diagnosis, or treatment recommendations. Any research involving SHLP-2 must comply with all applicable local, state, and federal regulations. BioInfinity Lab makes no claims regarding the safety or efficacy of SHLP-2 for any purpose. Consult qualified professionals for any research applications.